Which muscles are targeted by OMT to address hypertonicity that contributes to parkinsonian posturing?

Which muscles are targeted by OMT to address hypertonicity that contributes to parkinsonian posturing?

• A. Psoas and sternocleidomastoid
• B. Latissimus dorsi and trapezius
• C. Masseter and temporalis
• D. Diaphragm and intercostals

Answer: (A)

When aiming to lessen hypertonicity that drives parkinsonian posturing, focus is on axial and proximal muscles that set whole-body alignment. The psoas major, a deep hip flexor, pulls the pelvis into anterior tilt and encourages a forward-flexed spine. Chronic tightness here contributes to the stooped posture and reduced gait efficiency seen in Parkinson’s. Using osteopathic techniques to release or balance the psoas can help restore pelvic orientation, reduce compensatory rigidity in the lower spine, and improve overall posture. The sternocleidomastoid is a key neck flexor and stabilizer. When hypertonic, it can lock the head and neck into a flexed or rotated position, perpetuating abnormal upper spine posture and neck rigidity. Addressing SCM tension helps restore more neutral head carriage and reduces the cascading effects of neck rigidity on the shoulders and trunk. Together, relaxing these two muscles targets the core contributors to abnormal posturing in Parkinson’s and supports broader improvements in mobility and balance. The other muscle groups listed are less directly linked to the primary postural patterns of parkinsonian rigidity.

When aiming to lessen hypertonicity that drives parkinsonian posturing, focus is on axial and proximal muscles that set whole-body alignment. The psoas major, a deep hip flexor, pulls the pelvis into anterior tilt and encourages a forward-flexed spine. Chronic tightness here contributes to the stooped posture and reduced gait efficiency seen in Parkinson’s. Using osteopathic techniques to release or balance the psoas can help restore pelvic orientation, reduce compensatory rigidity in the lower spine, and improve overall posture.

The sternocleidomastoid is a key neck flexor and stabilizer. When hypertonic, it can lock the head and neck into a flexed or rotated position, perpetuating abnormal upper spine posture and neck rigidity. Addressing SCM tension helps restore more neutral head carriage and reduces the cascading effects of neck rigidity on the shoulders and trunk.

Together, relaxing these two muscles targets the core contributors to abnormal posturing in Parkinson’s and supports broader improvements in mobility and balance. The other muscle groups listed are less directly linked to the primary postural patterns of parkinsonian rigidity.